Synapse Formation

An Experimental Study of Synapse's Formation and Function When Transplanting Neural Stem Cells into PNS to Delay the Denervated Muscle Atrophy

神经干细胞移植延缓失神经肌萎过程中突触形成及其功能的实验研究

The rate of synapse formation in L1 transfection group was ( 58.0 ± 11.5)%, it was higher than that of non-transfection group ( 36.7 ± 0.83)% or Mock-transfection group ( 39.2 ± 0.84)% ( P < 0.01).

L1转染组功能性突触的形成率为（58.0±11.5）%，也高于非转染组的（36.7±0.83）%或Mock-转染组的（39.2±0.84）%（P

Objective To evaluate the role of interstitial cells of Cajal and synapse formation in congenital hypertrophic pyloric stenosis ( CHPS).

目的研究先天性肥厚性幽门狭窄（CHPS）幽门卡哈尔间质细胞发育和突触形成情况。

The function requires accurate connections of the synapses among the neurons, which not only needs the synapse formation, but also relies on the neural transmitter releasing.

学习记忆功能需要神经元之间精确的突触联系，这不但依赖于神经元突触的形成，还依赖于神经递质的释放。

Objective To study the effect of astrocyte on synapse formation and the molecular mechanism.

目的观察星形胶质细胞（AST）分泌雌激素的规律，研究AST对大脑皮层神经元突触形成的影响及可能的分子机制。

Synapse Formation between Rat Spinal Motor Neurons and Major Pelvic Ganglion Neurons in Vitro

大鼠脊髓运动神经元与盆节神经元之间在体外突触形成的研究

Neuronal cell adhesion molecule L1 enhances neurite outgrowth and functional synapse formation

神经细胞黏附分子L1促进神经细胞突起和功能性突触的形成

During which, some structural protein may affect AMPA receptor distribution, synapse formation, and hence synaptic plasticity and memory reconsolidation.

在此过程中，某些结构蛋白能够影响AMPA受体的分布和突触的形成，因此影响突触的可塑性以及记忆再巩固。

Synapse Formation and Electricity Physiology of Adult Human Neural Stem Cells Following Transplantation

成人神经干细胞脑内移植后突触形成和电生理功能的实验研究

The membrane-bound axon guidance molecule Netrin-G2 is preferentially expressed in the central nervous system and plays a role in synapse formation and maintenance.

目的：Netrin-G2是一种主要在中枢神经系统表达的神经轴突导向因子，在轴突的形成及神经细胞的迁移中发挥作用。

The results indicate that Nogo-A expressed in neuron may have function ( s) in addition to the neurite growth inhibitory activity, and Nogo-A may play a role in the course of neuronal differentiation such as neurite growth and synapse formation.

以上结果提示NOgO－A在神经元中具有不同于抑制作用的其他功能，可能参与神经元突起生长、突触形成等分化过程。

In particular, under conditions of injury, such as trauma, ischemia and hypoxia, normal resting astrocytes are transformed into activated ones to participate in the regulation of synapse formation and neural plasticity.

当中枢神经系统发生损伤，如外伤、局部缺血和缺氧等，星形胶质细胞会由静息态进入活化态，即发生星形化，从而参与突触形成和神经可塑性的调节。

Recent studies shed new light on the molecular mechanisms of central synapse formation by discovering a group of cell adhesion molecules exerting potent synaptogenic effects.

近期的研究又有新的亮点，发现一组细胞黏附分子具有很强的突触发生作用，使中枢突触形成的分子机制更加明朗。

Results: Fujian Tablet could increase the expression of neurotropic factor and decrease that of inhibitory factor. It promoted proliferation, migration, differentiation of neural stem cells, axonal growth and synapse formation remarkably.

结果：复健片可增加神经生长营养因子表达，减少抑制因子表达，对神经元增殖、迁移、分化，轴突延伸、突触形成等有明显促进作用。

Objective: To develop the mechanistic model for the reorientation of T cell receptors during immunological synapse formation.

目的：建立T细胞受体（TCR）在免疫突触形成过程中作重定向（reorientation）运动的机制模型。

Alterations in the estrogenic milieu of the developing central nervous system ( CNS) influence critical aspects of cellular differentiation, including neurite extension and branching, synapse formation, myelination, the expression of neurotransmitters and neuropeptides, and cell death and survival.

中枢神经系统发育过程中雌激素环境的改变可以影响细胞分化的几个关键方面：包括轴突的延长和分支、突触形成、髓鞘形成、神经递质和神经肽类的表达、以及细胞的存活与凋亡。

Cholesterol is crucial for the development of nervous system, membrane transport, signal transduction, synapse formation.

胆固醇在神经系统发育，膜运输，信号转导，突触形成以及神经元发挥正常功能都有重要作用。

Reorientation of T cell receptors during immunological synapse formation: a vortex-driven model

免疫突触形成过程中T细胞受体的重定向运动：涡旋驱动模型